The Cause Of Hypersensitivity Reactions And Allergy

Millions of Canadians suffer from allergies and asthma.mycobacteria, fungi, parasites, and, often,
When your immune system identifies a normallymucocutaneous candidiasis. Undesirable consequences
harmless material as a threat, it can set offof delayed-type hypersensitivity (DTH) reactions
symptoms like sniffles, sneezes, rashes or breathinginclude illness such as contact dermatitis and allograft
problems.rejection.
Although allergies and colds have similar symptoms,Examples of DTH reactions are contact dermatitis
there are some telltale signs that can help you(eg, poison ivy rash), tuberculin skin test reactions,
differentiate between them. If you aren't suregranulomatous inflammation (eg, sarcoidosis, Crohn
whether you have allergies or just a cold, ask yourdisease), allograft rejection, graft versus host
health professional. Your pharmacist or doctor candisease, and autoimmune hypersensitivity reactions.
help determine the cause of your symptoms andOf note, the Rhus genus of plants, which includes
recommend appropriate treatment.poison ivy, poison oak, and poison sumac, all cause
Experts think that 1-2 percent of Canadians haveidentical rashes.
serious allergies to food, insects, medications, latex,The cellular events that result in delayed
and exercise. These allergies are potentiallyhypersensitivity reactions primarily involve T cells and
life-threatening emergencies that require immediatemacrophages. First, local immune and inflammatory
medical attention.responses at the site of foreign antigen up-regulate
A person experiencing a serious allergic reaction mayendothelial cell adhesion molecule expression,
have difficulty breathing, throat swelling, skin rednesspromoting the accumulation of leukocytes at the
or itching, and an irregular heartbeat or palpitations. Intissue site. The antigen is engulfed by macrophages
most cases, it is recommended that people with anand monocytes and is presented to a T cell that has
identified serious allergy wear a MedicAlert braceleta specific receptor for that antigen. Macrophages
and carry an EpiPen, which is an injection device thatsecrete interleukin (IL)1, IL-2, IL-6, and other
contains epinephrine.lymphokines. Cytotoxic T cells can also be activated.
A recent study showed that two out of threeThe recruited macrophages can form giant cells. The
parents were unsure of how to use their child'scharacteristic histologic appearance of the
EpiPen, and almost half did not even carry theirmacrophage T-cell infiltrate is a granuloma. This type
EpiPen with them at all times. If you or a familyof infiltrate in the tissue is called granulomatous
member have a serious allergy, talk to yourinflammation.
pharmacist or doctor, and make sure you know howSeveral variants of DTH exist, and their precise
to recognize all the signs of a potentiallypathophysiologic mechanisms are slightly different. For
life-threatening reaction so that you know exactlyexample, in contact hypersensitivity reactions, the
what to do if the time ever comes.epidermis is involved; in pulmonary tuberculosis (TB),
Delayed hypersensitivity reactions are inflammatorylung tissue is involved.
reactions initiated by mononuclear leukocytes. TheDelayed hypersensitivity reactions are normal
term delayed is used to differentiate a secondaryphysiological events. Anything that alters these
cellular response, which appears 48-72 hours afternormal events can lead to multiple opportunistic
antigen exposure, from an immediate hypersensitivityinfections. Immune deficiencies (congenital or
response, which generally appears within 12 minutesacquired) and immunosuppressive agents can alter
of an antigen challenge. These reactions are mediatedthis normal response.he cellular events that result in
by T cells and monocytes/macrophages rather thandelayed hypersensitivity reactions primarily involve T
by antibodies. They are also termed type IVcells and macrophages. First, local immune and
hypersensitivity reactions.inflammatory responses at the site of foreign antigen
Delayed hypersensitivity is a major mechanism ofup-regulate endothelial cell adhesion molecule
defense against various intracellular pathogens,expression, promoting the accumulation of leukocytes
including mycobacteria, fungi, and certain parasites,at the tissue site. The antigen is engulfed by
and it occurs in transplant rejection and tumormacrophages and monocytes and is presented to a
immunity. The central role of CD4+ T cells in delayedT cell that has a specific receptor for that antigen.
hypersensitivity manifests in patients with AIDS.Macrophages secrete IL-1, IL-2, IL-6, and other
Because of the loss of CD4+ cells, the host responselymphokines. Cytotoxic T cells can also be activated.
against intracellular pathogens such as MycobacteriumThe recruited macrophages can form giant cells. The
tuberculosis is markedly impaired. The bacteria arecharacteristic histologic appearance of the
engulfed by macrophages but are not killed.macrophage T-cell infiltrate is a granuloma. This type
If T-cell function is abnormal, the patient presentsof infiltrate in the tissue is called granulomatous
with opportunistic infections, including infection withinflammation.